Saturday, October 6, 2007
Nolvadex (Tamoxifen Citrate)
Nolvadex (Tamoxifen Citrate) Buy Nolvadex (Tamoxifen Citrate) Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects. It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body. "Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do? Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production! * High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes. Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary. It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent. Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last. A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices. *Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex. Reported Characteristics Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Friday, October 5, 2007
Nolvadex (Tamoxifen Citrate)
Nolvadex (Tamoxifen Citrate)
Buy Nolvadex (Tamoxifen Citrate)
Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects.
It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body.
"Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do?
Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production!
* High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes.
Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary.
It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent.
Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last.
A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices.
*Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex.
Reported Characteristics
Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Buy Nolvadex (Tamoxifen Citrate)
Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects.
It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body.
"Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do?
Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production!
* High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes.
Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary.
It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent.
Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last.
A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices.
*Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex.
Reported Characteristics
Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Wednesday, October 3, 2007
Deca-Durabolin
Deca-Durabolin Buy Deca-Durabolin Nandrolone Decanoate is a very anabolic and slightly androgenic form of 19-nortestosterone A very prominent positive nitrogen balance is realized with administration of this product. And since nandrolone promotes nitrogen storage, growthand repair in hard-trained muscles will bemore pronounced than normal. In addition, a noted effect by many athletes polled was that most of them experienced a joint healing effect during Deca cycles. Since aromatization was low, in 400-600-mg weekly dosages, and fatburning was high due to the high affinity to the androgen recepror that Deca has, anti-estrogens were not typically necessary to avoid gyno and other estrogen induced side effects. British Dragon's Nandrolone Decanoate 200mg/ml 10ml bottle. Quickly becoming a best seller according to many bodybuilders Commonly used doses for men were in the area of 400-600mg weekly, and dosages over 400 mgweekly caused more water retention, while women seemed to do well with 100mgs/week. In fact, women have consistently reported very respectable lean mass and strength gains at dosages of only 50-100 mg weekly. Virilizing effects usually were avoided by single weekly injections of 50-100 mg nandrolone decanoate. Men, however would need to stack Deca with testosterone in order to have more appreciable weight gains, and avoid sexula dysfunction. Unfortunately, this compound is detectable for up to a year and a half after administration, so drug tested athletes should avoid it. Reported Characteristics Pharmaceutical Name:Nandrolone Decanoate Chemical Name:19-nor-testosterone Cutting/Bulking:Both Anabolic Rating: 125 Active-Life: 14-16 days Drug Class: Anabolic/Androgenic steroid Average Reported Dosage: Men 400-600mg weekly, Women 50-100mg weekly Acne: Possible Water Retention: Some Decreases HPTA function: Severe High Blood Pressure: No. Aromatization: Low, converts to less active norestrogens Liver Toxic: No DHT Conversion: No, converts to NOR- DHT Noted Comments: Highly anabolic/moderate androgenic effects Average Price: 10/amp TRADE NAMES DECA DURABOLIN 250mg/ml (British Dragon) DECA-DURABOLIN 200mg/2ml vial (Norma Greece) DECA-DURABOLIN (Organon) ANABOLINE 50-MG/ML ANDROLONE- D 200 200-MG/ML DECA DURABOLIN 25-MG/ML DECA DURABOLIN 50-MG/ML DECA DURABOLIN "100" 100-MG/ML DECA DURABOLIN 200-MG/ML ELPIHORM 50MG/ML EXTRABOLINE 50MG/ML HYBOLIN DECANOATE 50,100MG/ML JEBOLAN 50MG/ML NANDROLONE DECANOATE 50,100, 200MG /ML NANDROBOLIC L.A. 100MG/ML NEO-DURABOLIC 100,200,/ML NUREZAN 50MG/ML RETABOLIL 25MG/ML RETABOLIL 50MG /ML DECANANDROLEN 200 200MG/ML STEROBOLIN 50MG/ML TURINABOL DEPOT 50MG/ML ANABOLICAN 25ML/ML 10, 50ML
Tuesday, October 2, 2007
Arimidex (Anastrozole)
Arimidex Anastrozole Arimidex (Anastrozole) is what we call an aromatase inhibitor (AI). In clinical use, it´s used to halt the progression of Breast Cancer in women. It works by blocking the aromatase enzyme, which is responsible for the production of estrogen. In athletics and bodybuilding, it is used as an ancillary compound to be added to a cycle of Anabolic Steroids. In this respect it is also used for its estrogen reducing properties, but it has the additional benefit of increasing testosterone levels, as we´ll see... Arimidex Side EffectsMany anabolic steroids aromatize (convert to estrogen via the aromatase enzyme), and this is responsible for many of the unwanted side effects found with anabolic steroid use (acne, gynocomastia, water-retention, etc...). In one study, both .5mg and 1mg doses of Arimidex were shown to decrease estrogen by roughly 50%. The 1mg/day dose also increased testosterone levels by 58% (1). In that same study, in both groups, LH and FSH also went up slightly. Take a look: Changes in testosterone and E2 concentrations in normal young men (15 22 yr old) before () and after 10 days of oral anastrozole at 0.5 and 1 mg.(1) This would seem to suggest that for use during a cycle, a dose of .5mgs/day would be sufficient to combat estrogen-related side effects. It is, however, important to remember that some estrogen is necessary to obtain optimal muscle growth. The lower estrogen levels provided by ´dex seems, anecdotally at least, to produce a more "hard" and "quality" look for bodybuilders who have experimented with it´s use in either a cutting or bulking cycle. I´d like to point out that the elevation in Testosterone provided by Arimidex is so large that it can be used as a "form" of testosterone replacement therapy for hypogonadal men (2). Clearly, this suggests its use in a post-cycle-therapy (as well as its previously discussed use within a cycle) to regain natural testosterone levels and full functioning of the HPTA (Hypothalamic-Testicular-Pituitary-Axis). Literature provided by the original maker of Anastrozole (Arimidex, produced by Zeneca Pharmaceuticals) states that stable blood plasma concentrations of the compound are achieved after a mere 7 consecutive 1mg daily doses. Also, Arimidex is just over 80% effective at inhibiting aromatase (3). Thus, if you want to take it for the entire duration of a cycle of anabolic steroids, you can simply start taking it on the same day you begin your cycle. Those are some pretty good numbers, huh? But can you use it for the entire duration of a cycle? Is it dangerous? Well, certainly reducing estrogen levels in your body is good from a body building point of view, as it reduces water-retention and the potential for gynocomastia (if there´s no estrogen in your body, you can´t get gyno, regardless of how much progesterone is floating around)(5). Luckily this stuff is very mild on blood lipids (cholesterol) and doesn´t affect them adversely (2), in the studies I´ve seen. Arimidex and CholestrolAs previously mentioned, those lowered estrogen levels could possibly (eventually) adversely affect your cholesterol and possibly even your immune function. I am, however, very comfortable recommending Arimidex for relatively long-term use. This should be the ancillary compound of choice for those on long and heavy cycles, especially since it also doesn´t inhibit igf like some other ancillary compounds (insulin-like-growth-factor is an important component of anabolism)(4). Price of ArimidexThough price of Arimidex will vary, this is one of the compounds I will caution the reader from buying in its legitimate pharmaceutical form. The price (up to $5/tab) is absurd, when you consider its availability from Underground Labs, as well as in research form, for less than 1/3rd of that. I´ve used both the tabs from an Underground Lab, as well as the liquid version from research-sites, and found the results from both to be exactly the same. References: J Clin Endocrinol Metab 2000 Jul;85(7):2370-7, "Estrogen Suppression in Males" Clin Endocrinol (Oxf). 2005 Feb;62(2):228-35. Arimidex Package insert J Steroid Biochem Mol Biol. 2002 Apr;80(4-5):411-8. Progesterone is not essential to the differentiative potential of mammary epithelium in the male mouse. Freeman, Topper. Endocrinology. 1978 Jul;103(1):186-92
Monday, October 1, 2007
Dianabol
Dianabol is one of the most highly effective mass building steroids ever created. It was, in fact, created specifically for athletes to use to improve performance (although it was claimed to have theraputic ability). It provideds highly impressive weight and strength gains for most users. Experiencing a 2-3kg weight gain per week is not unheard of, especially in novices. It must be noted that alot if this is water weight, and that with higher dosages gynecomastia (bitch tits), high blood pressure, and acne could occur. Dianabol (Methandrostenlone) when taken orally becomes active very rapidly, but only remains active for less than half a day. Often, for this reason, dosages were spread through the day. Ciba, the original makers of Dianabol claimed that 10 mgs of the product was enough for full androgen replacement in a man, and this dose increased androgen anabolic activity roughly 5 times over normal and provided a reduction in natural cortisol activity of between 50-70%. Despite this, many athletes take 50-100mgs daily. It would appear that over 50mgs/day, there is a point of diminishing return. Dianabol is most commonly found in tablet form Since Dianabol (often called D-bol by athletes) is a 17aa compound, side effects such as increased liver values (toxicity) are to be expected, although they generally return to normal quickly after the athlete stops taking the product. In addition, as previously noted, gyno, heavy water retention (and raised blood pressure as a result), and acne were all commonly reported side effects of D-bol use. Interestingly, some users note a sense of well being during use of this drug, which is quite pronounced. This is the exact opposite of Anadrol, which is noted for an "unwell" feeling in users. This product is also available in an injectable form from both British Dragon (in oil) as well as under the Reforvit-B name (which has the addition of B vitamins) which is suspended in water and is quite frankly a very painful injection, and not often used for this reason. TRADE NAMES NAPOSIM 5MG TABS METHANDO (Akrihin) METHANABOL (British Dragon) AVERBOL 25 injectable, 25mg/ml British Dragon ANABOL TABS 5MG TABS ANABOLIN 5MG TABS ANDOREDAN 5MG TABS BIONABOL 2,5MG TABS DIALONE 5MG TABS DIANABOL 5MG ENCEPHAN 5MG TABS METANABOL 1,5MG TABS METHANDROSTENOLONUM 5MG TABS NEROBOL 5MG TABS PRONABOL-5 5MG TABS STENOLON 1,5MG TABS TRENERGIC 5MG CAPS NAPOSIM 5MG TABS D-BOL 10MG CAPSVETERINARY: ANABOLIKUM 2.5% 25MG/ML;50ML METANDIOBOL 25MG/ML;50ML D-BOL INJECTION 25MG/MLReported Characteristics Pharmaceutical Name:Methandrostenolone Chemical Structure:17a-methyl-17b-hydroxy-1,4-androstadien-3-one Cutting/Bulking:Bulking Anabolic Rating:90-210 Active-Life: 6-8 hours (24 for injectable) Drug Class: Anabolic/androgenic steroid Average Reported Dosage: Men 25-50 mg daily Acne: Possible Water Retention: Yes High Blood Pressure: Aromatization: Yes Liver Toxic: Yes DHT conversion: No Decreases HPTA function: Inhibitory Average Price: .50cents-$1 per 5-10mg tab
Tuesday, September 25, 2007
Nolvadex (Tamoxifen Citrate)
Nolvadex (Tamoxifen Citrate)
Buy Nolvadex (Tamoxifen Citrate)
Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects.
It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body.
"Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do?
Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production!
* High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes.
Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary.
It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent.
Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last.
A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices.
*Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex.
Reported Characteristics
Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Buy Nolvadex (Tamoxifen Citrate)
Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects.
It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body.
"Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do?
Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production!
* High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes.
Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary.
It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent.
Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last.
A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices.
*Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex.
Reported Characteristics
Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Monday, September 24, 2007
Nolvadex (Tamoxifen Citrate)
Nolvadex (Tamoxifen Citrate)
Buy Nolvadex (Tamoxifen Citrate)
Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects.
It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body.
"Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do?
Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production!
* High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes.
Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary.
It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent.
Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last.
A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices.
*Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex.
Reported Characteristics
Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Buy Nolvadex (Tamoxifen Citrate)
Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects.
It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body.
"Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do?
Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production!
* High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes.
Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary.
It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent.
Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last.
A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices.
*Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex.
Reported Characteristics
Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Friday, September 21, 2007
Cytomel ( liothyronine sodium )
Cytomel ( liothyronine sodium )
Cytomel (liothyronine sodium)
Quick overview:
Drug Class: Synthetic thyroid hormone Average Dose: 25-100 mcg/dayComments: Significant suppression of Thyroid function during use
Cytomel is the popularly recognized brand name for the drug liothyronine sodium. This is not an anabolic steroid but a thyroid hormone. It is used medically to treat cases of thyroid insufficiency, obesity, certain metabolic disorders and fatigue. Specifically this drug is a pharmaceutical preparation of the natural thyroid hormone triiodothyronine (T-3). When administered, T3 increases the patient's metabolism. The result is an increased rate of cellular activity (noted by a more rapid utilization of carbohydrates, fats and proteins). Bodybuilders are particularly attracted to this drug for its ability to burn off body excess fat. Most often utilized during contest preparation, one can greatly decrease the amount of stored fat without being forced to severely restrict calories. To this end Cytomel is commonly used in conjunction with Clenbuterol and can produce extremely dramatic results. This combination has become very popular in recent years, no doubt responsible for many "ripped" on-stage physiques. It is also noted by many that when thyroid hormones are taken in conjunction with steroids, an increased anabolic effect can be seen (noticeably greater than if the steroids are used alone). This is likely due to faster utilization of proteins by the body, increasing the rate for new muscle accumulation.
Caution should be taken if one is considering using this drug. Cytomel comes with an extensive list of warnings and precautions which are not to be ignored. Side effects include, but are not limited to, heart palpitations, agitation, shortness of breath, irregular heartbeat, sweating, nausea, headaches, and psychic/metabolic disorders. It is a powerful hormone, and one that could potentially alter the normal functioning of the body if misused. When taking Cytomel, one must remember to increase the dosage slowly. Generally one 25mcg tablet is taken on the first day, and the dosage is thereafter increased by one tablet every three of four days for a maximum dosage of 100mcg. This will help the body adjust to the increased thyroid hormone, hopefully avoiding any sudden "shock" to the system. The daily dose should also be split evenly throughout the day, in an effort to keep blood levels steadier. Women are more sensitive to the side effects of Cytomel than men, and usually choose not to take no more than 50mcg daily.
It is important to stress that a cycle should last no longer than 6 weeks and it should never be halted abruptly. As slowly as the dosage was built up it should also be lowered, one tablet every 3-4 days. Taking Cytomel for too long and/or at too high a dosage can result in a permanent thyroid deficiency. After doing such, one might need to be treated with a drug like Cytomel for life. It is also a good idea to first consult your physician and have your thyroid function tested. An undiagnosed hyperfunction would not mix well with the added hormone. An athlete should also be sure never to purchase an injectable form of the drug. It is generally an emergency room product, much too powerful for athletic use. Since T-3 is the most powerful thyroid hormone athletes are using, this is generally not the starting point for a beginner. Before using such a powerful item, it is a good idea to become familiar with a weaker substance. The highly popular Triacana is very mild, allowing the user much more latitude (from severe side effects) than Cytomel. An in-between point is Synthroid (synthetic T-4), still weaker in action than Cytomel. Once the user is ready however, the fat burning effect of this hormone can be extremely dramatic.
Cytomel (liothyronine sodium)
Quick overview:
Drug Class: Synthetic thyroid hormone Average Dose: 25-100 mcg/dayComments: Significant suppression of Thyroid function during use
Cytomel is the popularly recognized brand name for the drug liothyronine sodium. This is not an anabolic steroid but a thyroid hormone. It is used medically to treat cases of thyroid insufficiency, obesity, certain metabolic disorders and fatigue. Specifically this drug is a pharmaceutical preparation of the natural thyroid hormone triiodothyronine (T-3). When administered, T3 increases the patient's metabolism. The result is an increased rate of cellular activity (noted by a more rapid utilization of carbohydrates, fats and proteins). Bodybuilders are particularly attracted to this drug for its ability to burn off body excess fat. Most often utilized during contest preparation, one can greatly decrease the amount of stored fat without being forced to severely restrict calories. To this end Cytomel is commonly used in conjunction with Clenbuterol and can produce extremely dramatic results. This combination has become very popular in recent years, no doubt responsible for many "ripped" on-stage physiques. It is also noted by many that when thyroid hormones are taken in conjunction with steroids, an increased anabolic effect can be seen (noticeably greater than if the steroids are used alone). This is likely due to faster utilization of proteins by the body, increasing the rate for new muscle accumulation.
Caution should be taken if one is considering using this drug. Cytomel comes with an extensive list of warnings and precautions which are not to be ignored. Side effects include, but are not limited to, heart palpitations, agitation, shortness of breath, irregular heartbeat, sweating, nausea, headaches, and psychic/metabolic disorders. It is a powerful hormone, and one that could potentially alter the normal functioning of the body if misused. When taking Cytomel, one must remember to increase the dosage slowly. Generally one 25mcg tablet is taken on the first day, and the dosage is thereafter increased by one tablet every three of four days for a maximum dosage of 100mcg. This will help the body adjust to the increased thyroid hormone, hopefully avoiding any sudden "shock" to the system. The daily dose should also be split evenly throughout the day, in an effort to keep blood levels steadier. Women are more sensitive to the side effects of Cytomel than men, and usually choose not to take no more than 50mcg daily.
It is important to stress that a cycle should last no longer than 6 weeks and it should never be halted abruptly. As slowly as the dosage was built up it should also be lowered, one tablet every 3-4 days. Taking Cytomel for too long and/or at too high a dosage can result in a permanent thyroid deficiency. After doing such, one might need to be treated with a drug like Cytomel for life. It is also a good idea to first consult your physician and have your thyroid function tested. An undiagnosed hyperfunction would not mix well with the added hormone. An athlete should also be sure never to purchase an injectable form of the drug. It is generally an emergency room product, much too powerful for athletic use. Since T-3 is the most powerful thyroid hormone athletes are using, this is generally not the starting point for a beginner. Before using such a powerful item, it is a good idea to become familiar with a weaker substance. The highly popular Triacana is very mild, allowing the user much more latitude (from severe side effects) than Cytomel. An in-between point is Synthroid (synthetic T-4), still weaker in action than Cytomel. Once the user is ready however, the fat burning effect of this hormone can be extremely dramatic.
Sunday, September 16, 2007
Human Growth Hormone (GH)
Human Growth Hormone (GH)
Buy Human Growth Hormone (GH) - Click Here
Human Growth Hormone (GH) has been a subject of debate since I was a kid.Natural (endogenous) GH is produced by the pituitary gland. Children produce 2 i.u."spurts" 4-7 times per day for 4-5 non-consecutive days during a 2-3 week period (duringgrowth spurts). That would equal 32-70 i.u. in only a 4-5 day span. A healthy adult'spituitary releases only 0.5-1.5 i.u. daily.
GH has quickly become a very popular product for athletes looking to reach the top level Until the mid 1980's, the only available form of exogenous (occurring outside thebody) GH was manufactured by taking the pituitary glands of dead corpses (like there area lot of "live" corpses running around?) and grinding them up. (I am not joking!). TheGH was then extracted and purified through a series of expensive procedures, packed andsold by prescription only for use by children suffering from stunted growth. About 1987,this form of GH was linked to a fatal brain disease called CREUTZFELD-JAKOBDISEASE, and removed from the market.
Enter Genetech and synthetic GH. The first synthetic GH was produced bygenetically altering transformed mouse cells /Ecoli. Natural GH has a 191 amino acidsequence where as the Protropin brand of GH produced by Genetech contains 192 aminoacids in its sequence. This may have the affect of causing the body to produce GH antibodieswhich deactivate the GH. Most synthetics now contain the normal 191 amino acidsequence, of which there are over a dozen available today.
GH has 3 effects any athlete desires: GH helps the body burn more adipose (fat)tissue by promoting the release of fatty acids to be used as energy. Normally at rest, thebody uses about an equal division of fat and carbohydrate calories. When the endocrinesystem senses a low circulatory level of glucose, the hypothalamus-pituitary-axis (HPA)reacts by releasing GH. The GH then triggers (through a series of enzymic/chemicalreactions) the release of fatty acids from adipose stores so metabolic energy requirementscan be met. This means exogenous GH administration has been well documented to dothe same.
GH has a very potent anabolic (protein synthesis/tissue building) effect. Inexerting anabolic effects, it can cause both hyperplasia (an increase in the number ofmuscle cells) and muscular hypertrophy (the enlargement of muscle cells). This change incell number is permanent and therefore means more cells to make bigger. GH also has ananabolic effect on soft tissues such as tendons, cartilage, and other connective tissue. Thismeans old injuries repair and strength increases due to stronger connective tissue… bothat an accelerated rate. It is a well known fact that GH is a powerful anti-catabolic agent(protein sparing). This effect has allowed modern bodybuilders to retain or even addsignificant lean mass tissue during calorie restricted periods (cutting phases) and becomethe shredded monsters of the new era.
When using GH many athletes were less than satisfied with their results. Mostlikely this was because they bought bogus GH. It was common to find GH for a hard-corepro bodybuilder cost about $35,000 or more, yearly. To test GH, most simply bought apregnancy test kit, mix a vial of (hopefully) GH and place a drop or two in the test area.If the test result was “pregnant”..they had been screwed. Most pregnancy test kits test forelevated gonadoltropins (which HCG is and GH is not). For those few, whose bodiesmanufactured GH anti-bodies (and GH failed to work for you) sorry about your luck. GH,used properly, has overwhelmingly been renowned as a genetic equalizer if used for thatpurpose.
Any polled athlete chose to use GH as a performance enhancing drug should havefirst understand at least the basics of its actions.
GH itself is not responsible for the majority of the effects seen from GH use.Actually GH is only a precursor to the so-called "good stuff". When GH passes throughthe liver, it is converted into INSULIN-LIKE GROWTH FACTORS (such as IGF-1).IGF-1 is a very active but unstable chemical, which is why the body waits until the lastsecond to make it naturally. The liver has a limited capacity to convert excess GH intoIGF-1 unless other chemical hormone levels are also elevated. Insulin, T-4/T-3 thyroidhormones, gonadotropins, androgens/anabolic hormones, and even estrogen andcorticosteroids all play an important role in the positive effects of GH. So they too wereoften exogenously elevated in what was considered “the correct ratios” by the largest ofthe self administering athletes. For the liver to convert high levels of GH to IGF-1 severaltimes a day and cause a high quality anabolic response, it was commonly noted that T-3thyroid hormone and insulin also needed be increased to accomplish the desired effect.
Triacana may be strong enough to increase thyroid activity, but Cytomel was consideredto be a better choice. Though some seemed to disagree, most emphatically believed that afast-acting insulin such as HUMULIN-R or Humalog was a better and safer choice ofexogenous insulin since they allowed better timing and have a much shorter effectiveperiod. This allowed the athletes to time insulin activity with the active period of GH atthe optimum absorption times such as upon waking and the first few hours after a workout.The result was less chance of fat accumulation and a heightened anabolic response.Since GH suppresses natural T-3 thyroid hormone release, the exogenous administrationof Triacana or Cytomel allowed for an elevated calorie intake that was utilized more forbuilding muscle and soft tissue than for adipose tissue storage. Many pro bodybuildersused Clenbuterol and/or ephedrine stacks with GH while dieting. Since Clenbuterol and Ephedrine both suppress natural insulin release, they usually stacked the GH and Clenbuterol /Ephedrine with a synthetic T-3 thyroid hormone and sometimes with insulinas well. The use of insulin was dependent upon whether it was a bulking or dieting phaseand depending on how their body responded to exogenous insulin use.
*I can not stress enough how dangerous insulin use can be. Comas and death are quitepossible if used wrong. If you wish to use it, please see a doctor for monitoring.AAS and/or Clenbuterol further enhance the anabolic effects of GH. From all buta few polled it was reported that excellent muscle mass gains resulted with the use of GHwhen other chosen hormone levels were also met (*also see "cycles") and one couldafford it. Also, beware of fake GH. It is more common than you may realize. It is anillegal drug and the black market is not always honest.
The question of dosage was a big one. For the purpose of stunted growthmanufacturers of GH (due to pituitary hyophysially caused stunted growth) state 0.3 i.u.weekly per LB of body weight. So for a 235 LB bodybuilder that would equal 70.5 i.u.weekly, meaning a daily total of about 10-i.u. However, even 2-3i.u. daily did producesome nice results over a 6-8 week period when the other reported hormone requirementswere met as well. Short high dosage burst cycles too were noted to create these results(which will be discussed later) by the more elite of those polled.*GH is medically administered intramuscularly or subcutaneously (under the skin).*When multiple injections were utilized, I personally noted better results withsubcutaneous administration.
*1-mg=2.7 i.u. of GH and some products are listed as such.With exception of those few whose insert states otherwise, the dry unmixed GHsubstance maybe stored at room temperature. Once the solution has been mixed with thedry GH powder, (SWIRLED, DO NOT SHAKEN) the mixture must be refrigerated andlasts for 24-hours before it begins to degrade. An interesting product has becomeavailable called DEPO-NUTROPIN that has an active-life of about a month. This wouldallow for fewer injections and a reduced price. Also, several patents run out thisyear so many overseas and less expensive GH preparation will soon be available in theU.S. by prescription only.
*Though no negative side effects were reported, the available literature does listseveral serious ones: Kidneys and heart enlargement, high blood pressure, diabetes, thyroidhormone deficiency, and acromegaly. For the most part, they are rare to say the least andusually would be from extreme dosages and lengths of cycles. But like most hormones, youjust do not know until it is a fact for you. Kind of scary, huh?When GH was utilized with an insulin protocol, it was considered important tospace injection periods between GH and insulin about an hour. Also if GH was utilizedonly twice daily, it was reported best to avoid natural high points of GH release such asfirst thing in the a.m., post-work out, and right before bed. This was if GH was utilizedwithout insulin.
Reported Characteristics
Active-Life: Varies upon injection method Drug Class: Growth Hormone/IGF-1 Precursor (For injection) Average Reported Dosage: 2-16 i.u. total daily (1mg=2.7 i.u) Acne: None Water Retention: Very rare High Blood Pressure: Very rare Liver Toxic: None Aromatization: None High Anabolic/No Androgenic Effects
Trade Names
CORPORMON 4 I.U GENOTONORM 4 I.U. GENOTROPIN 2,3,4,12 I.U. GENOTROPIN 16 I.U. GRORM 2,4 I.U HUMATROPE 4 I.U. HUMATROPE 5MG HUMATROPE 16 I.U. NORDITROPIN 12 I.U. NUTROPIN 10 MG PROTROPIN 10 MG.. SAIZEN 10 I.U. SOMATOHORM 4 I.U. ZOMACTORS 4,12 I.U.
Buy Human Growth Hormone (GH) - Click Here
Human Growth Hormone (GH) has been a subject of debate since I was a kid.Natural (endogenous) GH is produced by the pituitary gland. Children produce 2 i.u."spurts" 4-7 times per day for 4-5 non-consecutive days during a 2-3 week period (duringgrowth spurts). That would equal 32-70 i.u. in only a 4-5 day span. A healthy adult'spituitary releases only 0.5-1.5 i.u. daily.
GH has quickly become a very popular product for athletes looking to reach the top level Until the mid 1980's, the only available form of exogenous (occurring outside thebody) GH was manufactured by taking the pituitary glands of dead corpses (like there area lot of "live" corpses running around?) and grinding them up. (I am not joking!). TheGH was then extracted and purified through a series of expensive procedures, packed andsold by prescription only for use by children suffering from stunted growth. About 1987,this form of GH was linked to a fatal brain disease called CREUTZFELD-JAKOBDISEASE, and removed from the market.
Enter Genetech and synthetic GH. The first synthetic GH was produced bygenetically altering transformed mouse cells /Ecoli. Natural GH has a 191 amino acidsequence where as the Protropin brand of GH produced by Genetech contains 192 aminoacids in its sequence. This may have the affect of causing the body to produce GH antibodieswhich deactivate the GH. Most synthetics now contain the normal 191 amino acidsequence, of which there are over a dozen available today.
GH has 3 effects any athlete desires: GH helps the body burn more adipose (fat)tissue by promoting the release of fatty acids to be used as energy. Normally at rest, thebody uses about an equal division of fat and carbohydrate calories. When the endocrinesystem senses a low circulatory level of glucose, the hypothalamus-pituitary-axis (HPA)reacts by releasing GH. The GH then triggers (through a series of enzymic/chemicalreactions) the release of fatty acids from adipose stores so metabolic energy requirementscan be met. This means exogenous GH administration has been well documented to dothe same.
GH has a very potent anabolic (protein synthesis/tissue building) effect. Inexerting anabolic effects, it can cause both hyperplasia (an increase in the number ofmuscle cells) and muscular hypertrophy (the enlargement of muscle cells). This change incell number is permanent and therefore means more cells to make bigger. GH also has ananabolic effect on soft tissues such as tendons, cartilage, and other connective tissue. Thismeans old injuries repair and strength increases due to stronger connective tissue… bothat an accelerated rate. It is a well known fact that GH is a powerful anti-catabolic agent(protein sparing). This effect has allowed modern bodybuilders to retain or even addsignificant lean mass tissue during calorie restricted periods (cutting phases) and becomethe shredded monsters of the new era.
When using GH many athletes were less than satisfied with their results. Mostlikely this was because they bought bogus GH. It was common to find GH for a hard-corepro bodybuilder cost about $35,000 or more, yearly. To test GH, most simply bought apregnancy test kit, mix a vial of (hopefully) GH and place a drop or two in the test area.If the test result was “pregnant”..they had been screwed. Most pregnancy test kits test forelevated gonadoltropins (which HCG is and GH is not). For those few, whose bodiesmanufactured GH anti-bodies (and GH failed to work for you) sorry about your luck. GH,used properly, has overwhelmingly been renowned as a genetic equalizer if used for thatpurpose.
Any polled athlete chose to use GH as a performance enhancing drug should havefirst understand at least the basics of its actions.
GH itself is not responsible for the majority of the effects seen from GH use.Actually GH is only a precursor to the so-called "good stuff". When GH passes throughthe liver, it is converted into INSULIN-LIKE GROWTH FACTORS (such as IGF-1).IGF-1 is a very active but unstable chemical, which is why the body waits until the lastsecond to make it naturally. The liver has a limited capacity to convert excess GH intoIGF-1 unless other chemical hormone levels are also elevated. Insulin, T-4/T-3 thyroidhormones, gonadotropins, androgens/anabolic hormones, and even estrogen andcorticosteroids all play an important role in the positive effects of GH. So they too wereoften exogenously elevated in what was considered “the correct ratios” by the largest ofthe self administering athletes. For the liver to convert high levels of GH to IGF-1 severaltimes a day and cause a high quality anabolic response, it was commonly noted that T-3thyroid hormone and insulin also needed be increased to accomplish the desired effect.
Triacana may be strong enough to increase thyroid activity, but Cytomel was consideredto be a better choice. Though some seemed to disagree, most emphatically believed that afast-acting insulin such as HUMULIN-R or Humalog was a better and safer choice ofexogenous insulin since they allowed better timing and have a much shorter effectiveperiod. This allowed the athletes to time insulin activity with the active period of GH atthe optimum absorption times such as upon waking and the first few hours after a workout.The result was less chance of fat accumulation and a heightened anabolic response.Since GH suppresses natural T-3 thyroid hormone release, the exogenous administrationof Triacana or Cytomel allowed for an elevated calorie intake that was utilized more forbuilding muscle and soft tissue than for adipose tissue storage. Many pro bodybuildersused Clenbuterol and/or ephedrine stacks with GH while dieting. Since Clenbuterol and Ephedrine both suppress natural insulin release, they usually stacked the GH and Clenbuterol /Ephedrine with a synthetic T-3 thyroid hormone and sometimes with insulinas well. The use of insulin was dependent upon whether it was a bulking or dieting phaseand depending on how their body responded to exogenous insulin use.
*I can not stress enough how dangerous insulin use can be. Comas and death are quitepossible if used wrong. If you wish to use it, please see a doctor for monitoring.AAS and/or Clenbuterol further enhance the anabolic effects of GH. From all buta few polled it was reported that excellent muscle mass gains resulted with the use of GHwhen other chosen hormone levels were also met (*also see "cycles") and one couldafford it. Also, beware of fake GH. It is more common than you may realize. It is anillegal drug and the black market is not always honest.
The question of dosage was a big one. For the purpose of stunted growthmanufacturers of GH (due to pituitary hyophysially caused stunted growth) state 0.3 i.u.weekly per LB of body weight. So for a 235 LB bodybuilder that would equal 70.5 i.u.weekly, meaning a daily total of about 10-i.u. However, even 2-3i.u. daily did producesome nice results over a 6-8 week period when the other reported hormone requirementswere met as well. Short high dosage burst cycles too were noted to create these results(which will be discussed later) by the more elite of those polled.*GH is medically administered intramuscularly or subcutaneously (under the skin).*When multiple injections were utilized, I personally noted better results withsubcutaneous administration.
*1-mg=2.7 i.u. of GH and some products are listed as such.With exception of those few whose insert states otherwise, the dry unmixed GHsubstance maybe stored at room temperature. Once the solution has been mixed with thedry GH powder, (SWIRLED, DO NOT SHAKEN) the mixture must be refrigerated andlasts for 24-hours before it begins to degrade. An interesting product has becomeavailable called DEPO-NUTROPIN that has an active-life of about a month. This wouldallow for fewer injections and a reduced price. Also, several patents run out thisyear so many overseas and less expensive GH preparation will soon be available in theU.S. by prescription only.
*Though no negative side effects were reported, the available literature does listseveral serious ones: Kidneys and heart enlargement, high blood pressure, diabetes, thyroidhormone deficiency, and acromegaly. For the most part, they are rare to say the least andusually would be from extreme dosages and lengths of cycles. But like most hormones, youjust do not know until it is a fact for you. Kind of scary, huh?When GH was utilized with an insulin protocol, it was considered important tospace injection periods between GH and insulin about an hour. Also if GH was utilizedonly twice daily, it was reported best to avoid natural high points of GH release such asfirst thing in the a.m., post-work out, and right before bed. This was if GH was utilizedwithout insulin.
Reported Characteristics
Active-Life: Varies upon injection method Drug Class: Growth Hormone/IGF-1 Precursor (For injection) Average Reported Dosage: 2-16 i.u. total daily (1mg=2.7 i.u) Acne: None Water Retention: Very rare High Blood Pressure: Very rare Liver Toxic: None Aromatization: None High Anabolic/No Androgenic Effects
Trade Names
CORPORMON 4 I.U GENOTONORM 4 I.U. GENOTROPIN 2,3,4,12 I.U. GENOTROPIN 16 I.U. GRORM 2,4 I.U HUMATROPE 4 I.U. HUMATROPE 5MG HUMATROPE 16 I.U. NORDITROPIN 12 I.U. NUTROPIN 10 MG PROTROPIN 10 MG.. SAIZEN 10 I.U. SOMATOHORM 4 I.U. ZOMACTORS 4,12 I.U.
Thursday, September 13, 2007
Masteron (Drostanolone Propionate)
Masteron (Drostanolone Propionate)
Buy Masteron (Drostanolone Propionate)
Masteron is a highly androgenic injectable steroid that is derived from DHT (dihydrotestosterone). DHT does not aromatize to estrogen (and in fact may combat estrogenic sides), and as a result, there was no noted water retention during administration nor gynocomastia. Masteron is almost exclusively used during the last 4-5 weeks before a bodybuilding show at a dose of 100-mg every second day. Additionally, according to available literature, Masteron is not much of a mass drug, and it's always used for cutting, from what I've seen reported. Masteron has a receptor binding ability above that of testosterone, due to it's being DHT-derived, which should impart lypolytic (fat-burning) effects above that of testosterone, and also give it a nice strength building component.
Pictured above is British Dragon's MASTABOL
Masteron can bind to Sex-Hormone-Binding-Globulin, which prevents AAS from merging with their receptors. Masteron can stop this from happening. This may mean that it enhances cycles, by letting other steroids work more effectively. Proviron, which is often called "oral masteron" (*correctly) does this very well also. Usually, Masteron would be stacked with Testosterone Propionate and possibly Trenbolone Acetate, which would be administered all at once, every second or third day in the same injection.
Reported Characteristics
Chemical Name:Drostanolone Pharmaceutical Name:2-alpha-methyl-androstan-3-one-17beta-ol Cutting/Bulking:Cutting Anabolic Rating: 62 Active Life: 2-3 days Drug Class: Androgenic/Anabolic steroid (For injection) Average Reported Dosage: Men 300-500-mg weekly Women 100-350mg weekly Acne: Yes Water Retention: None High Blood Pressure: Rare Liver Toxic: None Aromatization: None Noted Comments: High Androgenic/Moderate Anabolic/Moderate anti-estrogenic DHT Conversion: DHT-derived Decreases HPTA Function: Low if any
Trade Names:
MASTABOL 100MG/ML (British Dragon) MASTERIL 100-MG/2-ML MASTERON 100-MG/2-ML
Buy Masteron (Drostanolone Propionate)
Masteron is a highly androgenic injectable steroid that is derived from DHT (dihydrotestosterone). DHT does not aromatize to estrogen (and in fact may combat estrogenic sides), and as a result, there was no noted water retention during administration nor gynocomastia. Masteron is almost exclusively used during the last 4-5 weeks before a bodybuilding show at a dose of 100-mg every second day. Additionally, according to available literature, Masteron is not much of a mass drug, and it's always used for cutting, from what I've seen reported. Masteron has a receptor binding ability above that of testosterone, due to it's being DHT-derived, which should impart lypolytic (fat-burning) effects above that of testosterone, and also give it a nice strength building component.
Pictured above is British Dragon's MASTABOL
Masteron can bind to Sex-Hormone-Binding-Globulin, which prevents AAS from merging with their receptors. Masteron can stop this from happening. This may mean that it enhances cycles, by letting other steroids work more effectively. Proviron, which is often called "oral masteron" (*correctly) does this very well also. Usually, Masteron would be stacked with Testosterone Propionate and possibly Trenbolone Acetate, which would be administered all at once, every second or third day in the same injection.
Reported Characteristics
Chemical Name:Drostanolone Pharmaceutical Name:2-alpha-methyl-androstan-3-one-17beta-ol Cutting/Bulking:Cutting Anabolic Rating: 62 Active Life: 2-3 days Drug Class: Androgenic/Anabolic steroid (For injection) Average Reported Dosage: Men 300-500-mg weekly Women 100-350mg weekly Acne: Yes Water Retention: None High Blood Pressure: Rare Liver Toxic: None Aromatization: None Noted Comments: High Androgenic/Moderate Anabolic/Moderate anti-estrogenic DHT Conversion: DHT-derived Decreases HPTA Function: Low if any
Trade Names:
MASTABOL 100MG/ML (British Dragon) MASTERIL 100-MG/2-ML MASTERON 100-MG/2-ML
Sunday, September 9, 2007
Nolvadex (Tamoxifen Citrate)
Nolvadex (Tamoxifen Citrate)
Buy Nolvadex (Tamoxifen Citrate)
Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects.
It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body.
"Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do?
Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production!
* High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes.
Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary.
It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent.
Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last.
A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices.
*Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex.
Reported Characteristics
Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Buy Nolvadex (Tamoxifen Citrate)
Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects.
It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body.
"Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do?
Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production!
* High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes.
Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary.
It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent.
Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last.
A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices.
*Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex.
Reported Characteristics
Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Friday, September 7, 2007
Masteron (Drostanolone Propionate)
Masteron (Drostanolone Propionate)
Buy Masteron (Drostanolone Propionate)
Masteron is a highly androgenic injectable steroid that is derived from DHT (dihydrotestosterone). DHT does not aromatize to estrogen (and in fact may combat estrogenic sides), and as a result, there was no noted water retention during administration nor gynocomastia. Masteron is almost exclusively used during the last 4-5 weeks before a bodybuilding show at a dose of 100-mg every second day. Additionally, according to available literature, Masteron is not much of a mass drug, and it's always used for cutting, from what I've seen reported. Masteron has a receptor binding ability above that of testosterone, due to it's being DHT-derived, which should impart lypolytic (fat-burning) effects above that of testosterone, and also give it a nice strength building component.
Pictured above is British Dragon's MASTABOL
Masteron can bind to Sex-Hormone-Binding-Globulin, which prevents AAS from merging with their receptors. Masteron can stop this from happening. This may mean that it enhances cycles, by letting other steroids work more effectively. Proviron, which is often called "oral masteron" (*correctly) does this very well also. Usually, Masteron would be stacked with Testosterone Propionate and possibly Trenbolone Acetate, which would be administered all at once, every second or third day in the same injection.
Reported Characteristics
Chemical Name:Drostanolone Pharmaceutical Name:2-alpha-methyl-androstan-3-one-17beta-ol Cutting/Bulking:Cutting Anabolic Rating: 62 Active Life: 2-3 days Drug Class: Androgenic/Anabolic steroid (For injection) Average Reported Dosage: Men 300-500-mg weekly Women 100-350mg weekly Acne: Yes Water Retention: None High Blood Pressure: Rare Liver Toxic: None Aromatization: None Noted Comments: High Androgenic/Moderate Anabolic/Moderate anti-estrogenic DHT Conversion: DHT-derived Decreases HPTA Function: Low if any
Trade Names:
MASTABOL 100MG/ML (British Dragon) MASTERIL 100-MG/2-ML MASTERON 100-MG/2-ML
Buy Masteron (Drostanolone Propionate)
Masteron is a highly androgenic injectable steroid that is derived from DHT (dihydrotestosterone). DHT does not aromatize to estrogen (and in fact may combat estrogenic sides), and as a result, there was no noted water retention during administration nor gynocomastia. Masteron is almost exclusively used during the last 4-5 weeks before a bodybuilding show at a dose of 100-mg every second day. Additionally, according to available literature, Masteron is not much of a mass drug, and it's always used for cutting, from what I've seen reported. Masteron has a receptor binding ability above that of testosterone, due to it's being DHT-derived, which should impart lypolytic (fat-burning) effects above that of testosterone, and also give it a nice strength building component.
Pictured above is British Dragon's MASTABOL
Masteron can bind to Sex-Hormone-Binding-Globulin, which prevents AAS from merging with their receptors. Masteron can stop this from happening. This may mean that it enhances cycles, by letting other steroids work more effectively. Proviron, which is often called "oral masteron" (*correctly) does this very well also. Usually, Masteron would be stacked with Testosterone Propionate and possibly Trenbolone Acetate, which would be administered all at once, every second or third day in the same injection.
Reported Characteristics
Chemical Name:Drostanolone Pharmaceutical Name:2-alpha-methyl-androstan-3-one-17beta-ol Cutting/Bulking:Cutting Anabolic Rating: 62 Active Life: 2-3 days Drug Class: Androgenic/Anabolic steroid (For injection) Average Reported Dosage: Men 300-500-mg weekly Women 100-350mg weekly Acne: Yes Water Retention: None High Blood Pressure: Rare Liver Toxic: None Aromatization: None Noted Comments: High Androgenic/Moderate Anabolic/Moderate anti-estrogenic DHT Conversion: DHT-derived Decreases HPTA Function: Low if any
Trade Names:
MASTABOL 100MG/ML (British Dragon) MASTERIL 100-MG/2-ML MASTERON 100-MG/2-ML
Masteron (Drostanolone Propionate)
Masteron (Drostanolone Propionate)
Buy Masteron (Drostanolone Propionate)
Masteron is a highly androgenic injectable steroid that is derived from DHT (dihydrotestosterone). DHT does not aromatize to estrogen (and in fact may combat estrogenic sides), and as a result, there was no noted water retention during administration nor gynocomastia. Masteron is almost exclusively used during the last 4-5 weeks before a bodybuilding show at a dose of 100-mg every second day. Additionally, according to available literature, Masteron is not much of a mass drug, and it's always used for cutting, from what I've seen reported. Masteron has a receptor binding ability above that of testosterone, due to it's being DHT-derived, which should impart lypolytic (fat-burning) effects above that of testosterone, and also give it a nice strength building component.
Pictured above is British Dragon's MASTABOL
Masteron can bind to Sex-Hormone-Binding-Globulin, which prevents AAS from merging with their receptors. Masteron can stop this from happening. This may mean that it enhances cycles, by letting other steroids work more effectively. Proviron, which is often called "oral masteron" (*correctly) does this very well also. Usually, Masteron would be stacked with Testosterone Propionate and possibly Trenbolone Acetate, which would be administered all at once, every second or third day in the same injection.
Reported Characteristics
Chemical Name:Drostanolone Pharmaceutical Name:2-alpha-methyl-androstan-3-one-17beta-ol Cutting/Bulking:Cutting Anabolic Rating: 62 Active Life: 2-3 days Drug Class: Androgenic/Anabolic steroid (For injection) Average Reported Dosage: Men 300-500-mg weekly Women 100-350mg weekly Acne: Yes Water Retention: None High Blood Pressure: Rare Liver Toxic: None Aromatization: None Noted Comments: High Androgenic/Moderate Anabolic/Moderate anti-estrogenic DHT Conversion: DHT-derived Decreases HPTA Function: Low if any
Trade Names:
MASTABOL 100MG/ML (British Dragon) MASTERIL 100-MG/2-ML MASTERON 100-MG/2-ML
Buy Masteron (Drostanolone Propionate)
Masteron is a highly androgenic injectable steroid that is derived from DHT (dihydrotestosterone). DHT does not aromatize to estrogen (and in fact may combat estrogenic sides), and as a result, there was no noted water retention during administration nor gynocomastia. Masteron is almost exclusively used during the last 4-5 weeks before a bodybuilding show at a dose of 100-mg every second day. Additionally, according to available literature, Masteron is not much of a mass drug, and it's always used for cutting, from what I've seen reported. Masteron has a receptor binding ability above that of testosterone, due to it's being DHT-derived, which should impart lypolytic (fat-burning) effects above that of testosterone, and also give it a nice strength building component.
Pictured above is British Dragon's MASTABOL
Masteron can bind to Sex-Hormone-Binding-Globulin, which prevents AAS from merging with their receptors. Masteron can stop this from happening. This may mean that it enhances cycles, by letting other steroids work more effectively. Proviron, which is often called "oral masteron" (*correctly) does this very well also. Usually, Masteron would be stacked with Testosterone Propionate and possibly Trenbolone Acetate, which would be administered all at once, every second or third day in the same injection.
Reported Characteristics
Chemical Name:Drostanolone Pharmaceutical Name:2-alpha-methyl-androstan-3-one-17beta-ol Cutting/Bulking:Cutting Anabolic Rating: 62 Active Life: 2-3 days Drug Class: Androgenic/Anabolic steroid (For injection) Average Reported Dosage: Men 300-500-mg weekly Women 100-350mg weekly Acne: Yes Water Retention: None High Blood Pressure: Rare Liver Toxic: None Aromatization: None Noted Comments: High Androgenic/Moderate Anabolic/Moderate anti-estrogenic DHT Conversion: DHT-derived Decreases HPTA Function: Low if any
Trade Names:
MASTABOL 100MG/ML (British Dragon) MASTERIL 100-MG/2-ML MASTERON 100-MG/2-ML
Tuesday, September 4, 2007
Methandrostenolone (Dianabol)
Methandrostenolone (Dianabol) is an anabolic steroid originally developed by John Ziegler and released in the US in 1956 by Ciba. It was used as an aid to muscle growth by bodybuilders until its ban by the FDA under the Controlled Substances Act. Despite this, methandrostenolone continues to be produced in countries such as Mexico under the trade name Reforvit-b, and is being manufactured in Russia, as well as Thailand, and subsequently is still seen on the United States black market. Production in most of Western Europe and the United States has ceased. Several successful athletes and professional bodybuilders have come forward and admitted long-term methandrostenolone use before the drug was banned, including Arnold Schwarzenegger [1] and Sergio Oliva [2]. Despite its illegality many athletes continue to use the drug for the muscle mass gains it can cause. Methandrostenolone does not react strongly with the androgen receptor, instead relying on activity not mediated by the receptor for its effects. These include dramatic increases in protein synthesis, glycogenolysis, and muscle strength over a short space of time. However, due to its mode of action, it decreases the rate of cell respiration and decreases production of red blood cells. In high doses (30 mg or more per day), side effects such as gynaecomastia, high blood pressure, acne and male pattern baldness may begin to occur. The drug causes severe masculinising effects in women even at low doses. In addition, it is metabolized into estradiol by aromatase. This means that without the administration of aromatase inhibitors such as Anastrozole or Aminoglutethimide, estrogenic effects will appear over time in men. Many users will combat the estrogenic side effects with Nolvadex or Clomid. In addition, as with other 17α-alkylated steroids, the use of methandrostenolone over extended periods of time can result in liver damage without appropriate care. In the early 1960s, doctors commonly prescribed a tablet per day for women as a tonic. This use was quickly discontinued upon discovery of the heavily masculinising effects of methandrostenolone. However, despite the lack of any known therapeutic applications, the drug remained legal until the early 1990s. The ban by the FDA was not completely successful in eliminating its use by bodybuilders, and methandrostenolone continues to be used illegally to this day, typically being stacked (combined) with drugs that react strongly with the androgen receptor, such as Oxandrolone, in order to increase the overall effectiveness of steroid use. The 17α-methylation of the steroid does allow it to pass through the liver without being broken down (hence causing the aforementioned damage to the liver) allowing it to be taken orally. It also has the effect of decreasing the steroid's affinity for sex hormone binding globulin, a protein that de-activates steroid molecules and prevents them from further reactions with the body. As a result, methandrostenolone is significantly more active than an equivalent quantity of testosterone, resulting in rapid growth of muscle tissue. However, the concomitant elevation in estrogen levels - a result of the aromatization of methandrostenolone - results in significant water retention. This gives the appearance of great gains in mass and strength, which prove to be temporary once the steroid is discontinued and water weight drops. Because of this, it is often used by bodybuilders only at the start of a "steroid cycle", to facilitate rapid strength increases and the appearance of great size, while compounds such as testosterone or nandrolone with long acting esters build up in the body to an appreciable amount capable of supporting anabolic function on their own.
Monday, September 3, 2007
Muscle-bound Hollywood superstar Sylvester Stallone was fined yesterday for bringing illegal human growth hormones
SYDNEY: Muscle-bound Hollywood superstar Sylvester Stallone was fined yesterday for bringing illegal human growth hormones that he said made him “feel and look good” into Australia.The 60-year-old star of the blockbuster Rocky boxing movies had faced a maximum fine of A$22,000, but the magistrate ordered him to pay just A$2,975 (US$2,452) because the actor had shown remorse.But Deputy Chief Magistrate Paul Cloran also ordered Stallone, who was not in the Sydney court where he was sentenced, to pay the prosecution costs of A$10,000, taking his total bill to just under A$13,000.Stallone last week apologised to the court for bringing 52 vials of a banned human growth hormone and testosterone, a male hormone used to improve muscle mass, into Australia when he came here to promote his latest Rocky movie.“There is no suggestion that the substances were being used for anything other than cosmetic or therapeutic purposes,” Cloran said.“Mr Stallone made an error of judgment and he has now done all he can to remedy the situation. He has shown contrition. He has expressed his remorse.”But Stallone, in an interview recorded in Hollywood at the weekend, suggested he had only pleaded guilty and accepted a conviction last week in order to make the case go away.“I didn’t do anything illegally but they have rules down there,” Stallone said in an interview with Australia’s Channel Nine. “Literally, I didn’t want to sit around and fight it so sometimes you just go, ‘fine, whatever you say’.”The one-time ‘Italian Stallion’ said earlier in documents he had been taking the substances under medical supervision for years to treat a medical condition, which was not revealed in court, and did not know they were illegal.But the magistrate said Stallone had failed to prove he had a valid prescription for the human growth hormone, Jintropin, which was found in his luggage at Sydney airport.Jintropin is not legally available for retail use in the US and therefore could not have been prescribed, the magistrate said, also referring to an interview the actor had with Australian customs officers.During that encounter, Stallone told officers that as one got older, the pituitary gland slowed, making one feel older. “This stuff gives your body a boost and you feel and look good,” Stallone said. “Doing Rambo is hard work and I am going to be in Burma (Myanmar) for a while. Where do you think I am going to get this stuff in Burma?”“I will not be without these. I cannot be without these.” Human growth hormone occurs naturally but can also be made in synthetic form to boost muscle mass.Stallone last week pleaded guilty to importing the banned growth hormone, but said in a written statement he had made a “terrible mistake.”“Never ever was it my intention to breach the laws and I realise that I should have properly informed myself about your customs,” Stallone said.His lawyer said Stallone, who shot to global fame in the 1976 movie Rocky, had been ignorant of Australian law and was “extremely mortified” over the incident that overshadowed the launch of his latest film here.But prosecutor John Agius told the court that the Hollywood icon had tried to “cover up” the fact that he had not declared four vials of testosterone by throwing them out of his hotel window as police arrived to search his room.Cloran said while Stallone had tried to deceive customs officers about the hormone, he had shown contrition.Stallone was just 29 when he made Rocky, which took in nearly US$450mn at the worldwide box office, but was twice that age last year when he made Rocky Balboa, the sixth film in the series.“Just because people get older doesn’t mean they abandon their dream or their ability to want to do something, so Rocky is symbolic of still wanting to participate,” Stallone told reporters recently. – AFP
Friday, August 31, 2007
Equipoise
Equipoise Buy Equipoise Equipoise (Boldenone Undeclynate), or "EQ" is one of the most widely used compounds by athletes today. Even though it is a vetrinary steroid, the quality of production is quite high due to its use in rather expensive animals. It is one of the safest and most effective steroids used today, by athletes, power lifters and bodybuilders. It can effect slow and continuous muscle mass and strength gains over a prolonged period, and generally cycles with this drug tend to be quite long because of this fact. Gains on a cutting cycle including Eq tend to be high quality lean mass, while bulking cycle gains with Eq aren't as "clean" but still have a very nice quality about them. This could be due to Eq's resistence to being converted to Estrogen, as well as DHT (technically, it converts to Dihydroboldenone). Equipoise has become much more popular in recent years since dosages hit 200mg/ml Men get good results from a 300-600mg weekly total. In the past, this included utilizing an every other day (EOD) injection schedule, because Eq was only available in very low concentrations (50-100mgs/ml). Recently, companies like British Dragon have begun offering Boldenone in a much higher concentration (200mgs/ml) making injections much easier. Women have made excellent progress with dosages of 50-100mg total weekly doses, and this usually means a 1 injections weekly schedule. Women as a rule, seem to prefer less scar-tissue build up than men are comfortable with. In this dose range, women almost never report virilizing side effects, and usually only experienced an elevated libido. Increased hair growth on face and legs is, of course, possible (though not probable).. Liver toxicity is not an issue with Boldenone, and it can be used for long times on either a bulking or cutting cycle, very safely. It has a reputation for appetite and red blood cell production stimulating effects, and this makes it very nice for a bulking cycle. Obviously with increased red blood cell count an increase in oxygen transport was also realized, as was improved nutrient transport, making less food become more efficient...a very nice effect for a cutting cycle. Athletes generally stacked Boldenone with testosterone and Anadrol for mass cycles. Dianabol, due to it's close resemblance to Eq chemically, is not usually used interchangably with Anadrol in cycles containing EQ (a rare exception). Cutting, or pre-contest cycles including Boldenone often include Trenbolones, Winstrol. With the afforementioned drugs, an anti-estrogen is generally not necessary. Reported Characteristics Boldenone Undeclynate 1,4-androstadiene-3-one-17b-ol Cutting/Bulking:Both Anabolic Rating: 100 Active-Life: 7-9 days Drug Class: Anabolic /Androgenic steroid (for injection) Average Reported Dosage: Men 300-600mg weekly Women 50-100mg weekly Acne: Rare Water Retention: Low High Blood Pressure: Rare Aromatization: About 50% of testosterone Liver Toxic: No DHT Conversion: No (*converts to DHB) Decreases HPTA function: Yes Average Price: $150/10ml bottle Trade Names EQUIPOISE 200MG/ML (British Dragon) ULTRAGAN 100 100MG/ML BOLDEBAL-H 50MG/ML EQUIPOISE 25MG,50MG, /ML ULTRAGAN 50 50MG/ML GANABOL 25MG, 50MG/ML PACE 25MG/ML VENBOL 25MG/ML
Thursday, August 30, 2007
Nolvadex (Tamoxifen Citrate)
Nolvadex (Tamoxifen Citrate) Buy Nolvadex (Tamoxifen Citrate) Nolvadex is a drug commonly referred to as an anti-estrogen. This would suggestless or no estrogen is produced due to the drug's actions as in the case of Teslac. Actually,Nolvadex is an estrogen antagonist, meaning it competes with estrogen at estrogenreceptor- sites. This prevents the active estrogen from entering its receptor and creatingan estrogenic complex capable of activity. Since many AAS aromatize (covert toestrogen) to some degree, the control of feminizing side effects (males should payattention here) is important. Males normally have a very low estrogen level. During AAScycles, due to aromatization, estrogen levels rise considerably. This elevated estrogenlevel can cause feminizing side effects such as increased fat deposits, water retention, andgynecomastia (growth of breast gland tissue and painful tumors under the nipple). As arule, it is more the ratio of androgens-to-estrogens than the simple increase in estrogenthat actually initiates feminizing side effects. It is important that the reader realizes that Nolvadex does not decrease estrogenproduction and that it simply blocks estrogen receptors. For this reason the suddendiscontinuance of Nolvadex will allow the increased level of circulating estrogen tomerge with the newly freed receptors and do feminine things to the body. "Enter Proviron". At the end of a steroid cycle, the body's natural testosteroneproduction can be impaired. Due to the aromatization of the AAS estrogen levels aresignificantly higher than normal and Nolvadex only helps by blocking the estrogenreceptors. If an athlete abruptly ends an AAS protocol without regeneration of the HPTAunder these conditions, much of the hard earned gains would disappear due to estrogenbecoming the dominant hormone. So what did the boys (that didn’t want to be a girl) do? Proviron is an anti-estrogen (*See "Proviron" for more info) that helps to preventestrogen production while elevating androgen levels. During the last week of an AAScycle, some male bodybuilders began a HCG protocol (*See HCG) and administered 25-mg Proviron/10-20-mg Novladex 1-2 times daily. This was commonly noted to almostcompletely suppress post-cycle estrogen and its activity. Since Nolvadex increases thebody's own testosterone production, as does HCG, much of the cycle gains were retainedquite well. Nolvadex has a direct effect on the hypothalamus and therefore increases therelease of Gonadotropic hormones to a minor degree. (The hormones that tell the Leydigcells in the testes to produce androgens such as testosterone are refereed to asGonadotropics) Many added Clomid (*See Clomid) to their post-cycle stacks beginning6-10 days after HCG and continued for the average reported two week duration. In mostcases the result was athletes with normal (or above) sex drive and androgen production! * High dosage use of Nolvadex can inhibit natural testosterone production. This is due toinhibition of enzymes needed for testosterone production by the testes. Nolvadex was normally layered into any protocol utilizing high aromatizing steroids suchas testosterone, Dianabol, or those that are progesterone receptor stimulators such asAnadrol-50. Those who were prone to high fat deposits, water retention, and gynoconsistently reported inclusion of Nolvadex. Many are were to obtain excellent estrogenicactivity suppression with only 10-mg daily while others noted the need for as much as60-mg daily (20mg 3 times daily). The best results and guidelines were obtained bystarting low and increasing dosages only when necessary. It is important for the reader to realize that AAS must have some estrogen presentin order to achieve their full positive potential effectiveness and provide the bestcommonly desired results. This is why many AAS lose their anabolic qualities whencombined with anti-estrogens. It is also why Methandriol magnifies the effects of thesame AAS. Those who used high anabolic/moderate-low androgenic steroids such asnandrolones, Primobolan, or Winstrol, and did not combine them with high aromatizingsteroids (such as testosterone) often considered not using Nolvadex during cycles the bestchoice when increased mass was the primary intent. Women who used Nolvadex usually did so because it aids in fat loss due to lessestrogenic activity. I have yet to see a female compete whom was able to achieve trulycut legs with out it. Women athletes often combined 10-20mg of Nolvadex with 50-75mgProviron daily for the last few weeks of dieting. Due to availability of Clenbuterol,Proviron dosages were reported lower as of late, at least in female fitness competitors.Women should be aware that birth control is an estrogen and Novladex will block itseffectiveness. Women have note irregular menstrual cycles, weaker menstrual bleeding,and sometimes skip periods all together during Nolvadex use. I know several women whouse Nolvadex for this reason and can not say I disagree with their choice. After all, theuse of progestin type birth control as a means of regulating or even stopping menstruationis becoming accepted in the medical circles at last. A few athletes have experienced a paradox when using high dosages of Nolvadex.Instead of lowering estrogenic activity, it increased it. What happened was that theAdrenal glands went into over drive producing a pro-hormone called DHEA. DHEA isactually an adrenal androgen normally secreted in lower levels. As circulating levelsincreased enzymic factors came into play. Research shows DHEA readily converts intoandrostenedione, and to some extent, estrogens in males. (That sucks!) The femaleendocrine system usually favors testosterone production from converted DHEA orandrostenedione. The newly formed estrogen then overwhelmed the estrogen receptorsblocking the intended qualities of Novladex. In this case, Proviron, and especially Teslacwhere notably better choices. *Gyno that fails to react to these drugs normally must be removed by surgery. DHTderivatives can cause increases endogenous estrogen production also in some individuals.Cytadren was a commonly co-administered drug with Nolvadex. Reported Characteristics Active-Life: Less than 24 hours Drug Class: Anti-estrogen/estrogen antagonist (Oral) Average Reported Dosage: 10-30-mg daily Acne: None Water Retention: No High Blood Pressure: Rare (not normally attributed to the drug itself) Liver Toxic: Yes
Wednesday, August 29, 2007
Human Growth Hormone (GH)
Human Growth Hormone (GH)
Buy Human Growth Hormone (GH) - Click Here
Human Growth Hormone (GH) has been a subject of debate since I was a kid.Natural (endogenous) GH is produced by the pituitary gland. Children produce 2 i.u."spurts" 4-7 times per day for 4-5 non-consecutive days during a 2-3 week period (duringgrowth spurts). That would equal 32-70 i.u. in only a 4-5 day span. A healthy adult'spituitary releases only 0.5-1.5 i.u. daily.
GH has quickly become a very popular product for athletes looking to reach the top level Until the mid 1980's, the only available form of exogenous (occurring outside thebody) GH was manufactured by taking the pituitary glands of dead corpses (like there area lot of "live" corpses running around?) and grinding them up. (I am not joking!). TheGH was then extracted and purified through a series of expensive procedures, packed andsold by prescription only for use by children suffering from stunted growth. About 1987,this form of GH was linked to a fatal brain disease called CREUTZFELD-JAKOBDISEASE, and removed from the market.
Enter Genetech and synthetic GH. The first synthetic GH was produced bygenetically altering transformed mouse cells /Ecoli. Natural GH has a 191 amino acidsequence where as the Protropin brand of GH produced by Genetech contains 192 aminoacids in its sequence. This may have the affect of causing the body to produce GH antibodieswhich deactivate the GH. Most synthetics now contain the normal 191 amino acidsequence, of which there are over a dozen available today.
GH has 3 effects any athlete desires: GH helps the body burn more adipose (fat)tissue by promoting the release of fatty acids to be used as energy. Normally at rest, thebody uses about an equal division of fat and carbohydrate calories. When the endocrinesystem senses a low circulatory level of glucose, the hypothalamus-pituitary-axis (HPA)reacts by releasing GH. The GH then triggers (through a series of enzymic/chemicalreactions) the release of fatty acids from adipose stores so metabolic energy requirementscan be met. This means exogenous GH administration has been well documented to dothe same.
GH has a very potent anabolic (protein synthesis/tissue building) effect. Inexerting anabolic effects, it can cause both hyperplasia (an increase in the number ofmuscle cells) and muscular hypertrophy (the enlargement of muscle cells). This change incell number is permanent and therefore means more cells to make bigger. GH also has ananabolic effect on soft tissues such as tendons, cartilage, and other connective tissue. Thismeans old injuries repair and strength increases due to stronger connective tissue… bothat an accelerated rate. It is a well known fact that GH is a powerful anti-catabolic agent(protein sparing). This effect has allowed modern bodybuilders to retain or even addsignificant lean mass tissue during calorie restricted periods (cutting phases) and becomethe shredded monsters of the new era.
When using GH many athletes were less than satisfied with their results. Mostlikely this was because they bought bogus GH. It was common to find GH for a hard-corepro bodybuilder cost about $35,000 or more, yearly. To test GH, most simply bought apregnancy test kit, mix a vial of (hopefully) GH and place a drop or two in the test area.If the test result was “pregnant”..they had been screwed. Most pregnancy test kits test forelevated gonadoltropins (which HCG is and GH is not). For those few, whose bodiesmanufactured GH anti-bodies (and GH failed to work for you) sorry about your luck. GH,used properly, has overwhelmingly been renowned as a genetic equalizer if used for thatpurpose.
Any polled athlete chose to use GH as a performance enhancing drug should havefirst understand at least the basics of its actions.
GH itself is not responsible for the majority of the effects seen from GH use.Actually GH is only a precursor to the so-called "good stuff". When GH passes throughthe liver, it is converted into INSULIN-LIKE GROWTH FACTORS (such as IGF-1).IGF-1 is a very active but unstable chemical, which is why the body waits until the lastsecond to make it naturally. The liver has a limited capacity to convert excess GH intoIGF-1 unless other chemical hormone levels are also elevated. Insulin, T-4/T-3 thyroidhormones, gonadotropins, androgens/anabolic hormones, and even estrogen andcorticosteroids all play an important role in the positive effects of GH. So they too wereoften exogenously elevated in what was considered “the correct ratios” by the largest ofthe self administering athletes. For the liver to convert high levels of GH to IGF-1 severaltimes a day and cause a high quality anabolic response, it was commonly noted that T-3thyroid hormone and insulin also needed be increased to accomplish the desired effect.
Triacana may be strong enough to increase thyroid activity, but Cytomel was consideredto be a better choice. Though some seemed to disagree, most emphatically believed that afast-acting insulin such as HUMULIN-R or Humalog was a better and safer choice ofexogenous insulin since they allowed better timing and have a much shorter effectiveperiod. This allowed the athletes to time insulin activity with the active period of GH atthe optimum absorption times such as upon waking and the first few hours after a workout.The result was less chance of fat accumulation and a heightened anabolic response.Since GH suppresses natural T-3 thyroid hormone release, the exogenous administrationof Triacana or Cytomel allowed for an elevated calorie intake that was utilized more forbuilding muscle and soft tissue than for adipose tissue storage. Many pro bodybuildersused Clenbuterol and/or ephedrine stacks with GH while dieting. Since Clenbuterol and Ephedrine both suppress natural insulin release, they usually stacked the GH and Clenbuterol /Ephedrine with a synthetic T-3 thyroid hormone and sometimes with insulinas well. The use of insulin was dependent upon whether it was a bulking or dieting phaseand depending on how their body responded to exogenous insulin use.
*I can not stress enough how dangerous insulin use can be. Comas and death are quitepossible if used wrong. If you wish to use it, please see a doctor for monitoring.AAS and/or Clenbuterol further enhance the anabolic effects of GH. From all buta few polled it was reported that excellent muscle mass gains resulted with the use of GHwhen other chosen hormone levels were also met (*also see "cycles") and one couldafford it. Also, beware of fake GH. It is more common than you may realize. It is anillegal drug and the black market is not always honest.
The question of dosage was a big one. For the purpose of stunted growthmanufacturers of GH (due to pituitary hyophysially caused stunted growth) state 0.3 i.u.weekly per LB of body weight. So for a 235 LB bodybuilder that would equal 70.5 i.u.weekly, meaning a daily total of about 10-i.u. However, even 2-3i.u. daily did producesome nice results over a 6-8 week period when the other reported hormone requirementswere met as well. Short high dosage burst cycles too were noted to create these results(which will be discussed later) by the more elite of those polled.*GH is medically administered intramuscularly or subcutaneously (under the skin).*When multiple injections were utilized, I personally noted better results withsubcutaneous administration.
*1-mg=2.7 i.u. of GH and some products are listed as such.With exception of those few whose insert states otherwise, the dry unmixed GHsubstance maybe stored at room temperature. Once the solution has been mixed with thedry GH powder, (SWIRLED, DO NOT SHAKEN) the mixture must be refrigerated andlasts for 24-hours before it begins to degrade. An interesting product has becomeavailable called DEPO-NUTROPIN that has an active-life of about a month. This wouldallow for fewer injections and a reduced price. Also, several patents run out thisyear so many overseas and less expensive GH preparation will soon be available in theU.S. by prescription only.
*Though no negative side effects were reported, the available literature does listseveral serious ones: Kidneys and heart enlargement, high blood pressure, diabetes, thyroidhormone deficiency, and acromegaly. For the most part, they are rare to say the least andusually would be from extreme dosages and lengths of cycles. But like most hormones, youjust do not know until it is a fact for you. Kind of scary, huh?When GH was utilized with an insulin protocol, it was considered important tospace injection periods between GH and insulin about an hour. Also if GH was utilizedonly twice daily, it was reported best to avoid natural high points of GH release such asfirst thing in the a.m., post-work out, and right before bed. This was if GH was utilizedwithout insulin.
Reported Characteristics
Active-Life: Varies upon injection method Drug Class: Growth Hormone/IGF-1 Precursor (For injection) Average Reported Dosage: 2-16 i.u. total daily (1mg=2.7 i.u) Acne: None Water Retention: Very rare High Blood Pressure: Very rare Liver Toxic: None Aromatization: None High Anabolic/No Androgenic Effects
Trade Names
CORPORMON 4 I.U GENOTONORM 4 I.U. GENOTROPIN 2,3,4,12 I.U. GENOTROPIN 16 I.U. GRORM 2,4 I.U HUMATROPE 4 I.U. HUMATROPE 5MG HUMATROPE 16 I.U. NORDITROPIN 12 I.U. NUTROPIN 10 MG PROTROPIN 10 MG.. SAIZEN 10 I.U. SOMATOHORM 4 I.U. ZOMACTORS 4,12 I.U.
Buy Human Growth Hormone (GH) - Click Here
Human Growth Hormone (GH) has been a subject of debate since I was a kid.Natural (endogenous) GH is produced by the pituitary gland. Children produce 2 i.u."spurts" 4-7 times per day for 4-5 non-consecutive days during a 2-3 week period (duringgrowth spurts). That would equal 32-70 i.u. in only a 4-5 day span. A healthy adult'spituitary releases only 0.5-1.5 i.u. daily.
GH has quickly become a very popular product for athletes looking to reach the top level Until the mid 1980's, the only available form of exogenous (occurring outside thebody) GH was manufactured by taking the pituitary glands of dead corpses (like there area lot of "live" corpses running around?) and grinding them up. (I am not joking!). TheGH was then extracted and purified through a series of expensive procedures, packed andsold by prescription only for use by children suffering from stunted growth. About 1987,this form of GH was linked to a fatal brain disease called CREUTZFELD-JAKOBDISEASE, and removed from the market.
Enter Genetech and synthetic GH. The first synthetic GH was produced bygenetically altering transformed mouse cells /Ecoli. Natural GH has a 191 amino acidsequence where as the Protropin brand of GH produced by Genetech contains 192 aminoacids in its sequence. This may have the affect of causing the body to produce GH antibodieswhich deactivate the GH. Most synthetics now contain the normal 191 amino acidsequence, of which there are over a dozen available today.
GH has 3 effects any athlete desires: GH helps the body burn more adipose (fat)tissue by promoting the release of fatty acids to be used as energy. Normally at rest, thebody uses about an equal division of fat and carbohydrate calories. When the endocrinesystem senses a low circulatory level of glucose, the hypothalamus-pituitary-axis (HPA)reacts by releasing GH. The GH then triggers (through a series of enzymic/chemicalreactions) the release of fatty acids from adipose stores so metabolic energy requirementscan be met. This means exogenous GH administration has been well documented to dothe same.
GH has a very potent anabolic (protein synthesis/tissue building) effect. Inexerting anabolic effects, it can cause both hyperplasia (an increase in the number ofmuscle cells) and muscular hypertrophy (the enlargement of muscle cells). This change incell number is permanent and therefore means more cells to make bigger. GH also has ananabolic effect on soft tissues such as tendons, cartilage, and other connective tissue. Thismeans old injuries repair and strength increases due to stronger connective tissue… bothat an accelerated rate. It is a well known fact that GH is a powerful anti-catabolic agent(protein sparing). This effect has allowed modern bodybuilders to retain or even addsignificant lean mass tissue during calorie restricted periods (cutting phases) and becomethe shredded monsters of the new era.
When using GH many athletes were less than satisfied with their results. Mostlikely this was because they bought bogus GH. It was common to find GH for a hard-corepro bodybuilder cost about $35,000 or more, yearly. To test GH, most simply bought apregnancy test kit, mix a vial of (hopefully) GH and place a drop or two in the test area.If the test result was “pregnant”..they had been screwed. Most pregnancy test kits test forelevated gonadoltropins (which HCG is and GH is not). For those few, whose bodiesmanufactured GH anti-bodies (and GH failed to work for you) sorry about your luck. GH,used properly, has overwhelmingly been renowned as a genetic equalizer if used for thatpurpose.
Any polled athlete chose to use GH as a performance enhancing drug should havefirst understand at least the basics of its actions.
GH itself is not responsible for the majority of the effects seen from GH use.Actually GH is only a precursor to the so-called "good stuff". When GH passes throughthe liver, it is converted into INSULIN-LIKE GROWTH FACTORS (such as IGF-1).IGF-1 is a very active but unstable chemical, which is why the body waits until the lastsecond to make it naturally. The liver has a limited capacity to convert excess GH intoIGF-1 unless other chemical hormone levels are also elevated. Insulin, T-4/T-3 thyroidhormones, gonadotropins, androgens/anabolic hormones, and even estrogen andcorticosteroids all play an important role in the positive effects of GH. So they too wereoften exogenously elevated in what was considered “the correct ratios” by the largest ofthe self administering athletes. For the liver to convert high levels of GH to IGF-1 severaltimes a day and cause a high quality anabolic response, it was commonly noted that T-3thyroid hormone and insulin also needed be increased to accomplish the desired effect.
Triacana may be strong enough to increase thyroid activity, but Cytomel was consideredto be a better choice. Though some seemed to disagree, most emphatically believed that afast-acting insulin such as HUMULIN-R or Humalog was a better and safer choice ofexogenous insulin since they allowed better timing and have a much shorter effectiveperiod. This allowed the athletes to time insulin activity with the active period of GH atthe optimum absorption times such as upon waking and the first few hours after a workout.The result was less chance of fat accumulation and a heightened anabolic response.Since GH suppresses natural T-3 thyroid hormone release, the exogenous administrationof Triacana or Cytomel allowed for an elevated calorie intake that was utilized more forbuilding muscle and soft tissue than for adipose tissue storage. Many pro bodybuildersused Clenbuterol and/or ephedrine stacks with GH while dieting. Since Clenbuterol and Ephedrine both suppress natural insulin release, they usually stacked the GH and Clenbuterol /Ephedrine with a synthetic T-3 thyroid hormone and sometimes with insulinas well. The use of insulin was dependent upon whether it was a bulking or dieting phaseand depending on how their body responded to exogenous insulin use.
*I can not stress enough how dangerous insulin use can be. Comas and death are quitepossible if used wrong. If you wish to use it, please see a doctor for monitoring.AAS and/or Clenbuterol further enhance the anabolic effects of GH. From all buta few polled it was reported that excellent muscle mass gains resulted with the use of GHwhen other chosen hormone levels were also met (*also see "cycles") and one couldafford it. Also, beware of fake GH. It is more common than you may realize. It is anillegal drug and the black market is not always honest.
The question of dosage was a big one. For the purpose of stunted growthmanufacturers of GH (due to pituitary hyophysially caused stunted growth) state 0.3 i.u.weekly per LB of body weight. So for a 235 LB bodybuilder that would equal 70.5 i.u.weekly, meaning a daily total of about 10-i.u. However, even 2-3i.u. daily did producesome nice results over a 6-8 week period when the other reported hormone requirementswere met as well. Short high dosage burst cycles too were noted to create these results(which will be discussed later) by the more elite of those polled.*GH is medically administered intramuscularly or subcutaneously (under the skin).*When multiple injections were utilized, I personally noted better results withsubcutaneous administration.
*1-mg=2.7 i.u. of GH and some products are listed as such.With exception of those few whose insert states otherwise, the dry unmixed GHsubstance maybe stored at room temperature. Once the solution has been mixed with thedry GH powder, (SWIRLED, DO NOT SHAKEN) the mixture must be refrigerated andlasts for 24-hours before it begins to degrade. An interesting product has becomeavailable called DEPO-NUTROPIN that has an active-life of about a month. This wouldallow for fewer injections and a reduced price. Also, several patents run out thisyear so many overseas and less expensive GH preparation will soon be available in theU.S. by prescription only.
*Though no negative side effects were reported, the available literature does listseveral serious ones: Kidneys and heart enlargement, high blood pressure, diabetes, thyroidhormone deficiency, and acromegaly. For the most part, they are rare to say the least andusually would be from extreme dosages and lengths of cycles. But like most hormones, youjust do not know until it is a fact for you. Kind of scary, huh?When GH was utilized with an insulin protocol, it was considered important tospace injection periods between GH and insulin about an hour. Also if GH was utilizedonly twice daily, it was reported best to avoid natural high points of GH release such asfirst thing in the a.m., post-work out, and right before bed. This was if GH was utilizedwithout insulin.
Reported Characteristics
Active-Life: Varies upon injection method Drug Class: Growth Hormone/IGF-1 Precursor (For injection) Average Reported Dosage: 2-16 i.u. total daily (1mg=2.7 i.u) Acne: None Water Retention: Very rare High Blood Pressure: Very rare Liver Toxic: None Aromatization: None High Anabolic/No Androgenic Effects
Trade Names
CORPORMON 4 I.U GENOTONORM 4 I.U. GENOTROPIN 2,3,4,12 I.U. GENOTROPIN 16 I.U. GRORM 2,4 I.U HUMATROPE 4 I.U. HUMATROPE 5MG HUMATROPE 16 I.U. NORDITROPIN 12 I.U. NUTROPIN 10 MG PROTROPIN 10 MG.. SAIZEN 10 I.U. SOMATOHORM 4 I.U. ZOMACTORS 4,12 I.U.
Subscribe to:
Posts (Atom)
